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Other experimental studies in vivo [65,66] have shown that GSH molecular pathways are increased, although not always significantly, after sulforaphane, a cruciferous-derived compound, administration [65]

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NAD, nicotinamide adenine dinucleotide

Again, the loss of correlations in the pathway metabolite may be lost in patients due to illness-related, and also perhaps treatment influences, on 5-HIAA, since the TRP5-HIAA correlation is appears weakened in patients (Yao et al., 2010a)

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