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Damage-associated molecular patterns, such as high mobility group box 1 (HMGB1), initiate this phenotypic shift by engaging signaling pathways like toll-like receptor 4 (TLR4) and the receptor for advanced glycation end products (RAGE), which in turn activate downstream effectors, including nuclear factor kappa-B (NF-B) and the NOD-like receptor thermal protein domain associated protein 3 (NLRP3) inflammasome (Tastan and Heneka, 2024

Factors like aging, poor diet, chronic stress, infections, and exposure to pollutants can deplete GSH stores, making dietary support and supplementation essential strategies for maintaining optimal health and resilience against disease

2020;23(Sep):65971

The 2000s and 2010s brought substantial expansion of the GHK-Cu literature through transcriptomic studies