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The studies were searched using the keywords Retatrutide, Triple Agonist, LY3437943, Diabetes and Obesity

GLP-1 RAs suppress NLRP3 inflammasome activation through multiple mechanisms: inhibition of priming signals via reduced NF-B activity, attenuation of reactive oxygen species generation through enhanced mitochondrial function, induction of autophagy that removes damaged mitochondria serving as inflammasome triggers, and direct interference with NLRP3-ASC interaction through PKA-mediated phosphorylation [38,39]

Lipo B operates through an entirely different mechanism