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doi: 10.1210/en.2003-0007

Husein El Hadmed H, Castillo RF

On one hand, increased cytochrome C release caused by mitochondrial calcium overload inhibits the negative feedback effect of cytoplasmic Ca 2+ on IP3 receptors, while on the other, it activates caspase3 to cleave IP3 receptors, hence forming a vicious cycle of massive calcium release from the ER[28]

More specifically, the 10 residue sequence of the cleavage site at the nsp13/14 junction shown in Figure 2A, with the greatest overall similarity to the GPX1 active site sequence, is 100% identical in the 2003 and 2019 SARS coronaviruses (Figure S1)

Here, we highlight emerging senomorphics in clinical stages and their translational potential