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These findings indicate that even though ethanol may not be required for TNBS-induced damage, this vehicle enhanced the biological action of TNBS in agreement with previous results (Morris et al, 1989), presumably by acting as a barrier breaker and facilitating its interaction with cellular GSH stores (Table 1)

None have undergone the randomized, controlled, large-scale trials that would establish definitive efficacy and safety for sleep indications

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The formulation combines Glycyl-L-histidyl-L-lysine copper(II) (GHK-Cu), Body Protection Compound-157 (BPC-157), Thymosin Beta-4 derivative (TB500), and the naturally occurring alpha-MSH cleavage derivative, KPV