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IL-18 acts in synergy with IL-12 to stimulate T cells production of IFN-, which in turn stimulates synovial macrophages to produce TNF- and IL-1, leading to joint inflammation and cartilage destruction (79)

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To elucidate the underlying molecular mechanism, small interfering ribonucleic acid (siRNA) for GLP-1R and Dorsomorphin, a specific inhibitor of adenosine monophosphate-activated protein kinase (AMPK), were intracerebroventricularly injected 48 h before induction of SAH correspondingly