If p53 is mutated, unable to function as a transcriptional regulator, and unable to mediate apoptosis, cells can continue to divide without DNA repair, chromosomal damage may occur and magnify with each cell division, and neoplasia can ensue (see Figure 2)
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Such an increase in energy dissipation, reflected by higher DI 0 /RC, has also been reported under various abiotic stresses, including heat, salinity, and drought, as a photoprotective mechanism ( 0 /RC and F 0 /F M under elevated CS concentrations suggests that CS imposes photoinhibitory pressure on PSII, and the plant attempts to mitigate this through increased energy dissipation
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