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The study found that FOS/GOS supplementation inhibited the dysregulated expression of intestinal epithelial gap junction proteinsincluding Claudin-1, Claudin-15, Zonula occludens-1 (ZO-1), and Junctional adhesion molecule-Athereby mitigating barrier damage and reversing HFD-induced hyperpermeability
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In decompensated patients, intestinal barrier disruption reaches an extreme, accompanied by severe microbial abnormalities and significant impairment of the intestinal physical, immune, and vascular barriers
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Nuez-AnitaREArroyo-HelgueraOCajero-JurezMLpez-BojorquezLAcevesC
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Clinical guidelines, as the post describes them, favor risk stratification with close monitoring over automatic exclusion
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McDonnell, G
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